Primary Faculty

Shanna Hamilton, PhD

BSc Biochemistry, Swansea University, 2012
PhD Medicine/Biophysics, Cardiff University, 2018
Postdoctoral Training in Cardiovascular Physiology, Brown University, 2018-2019
Postdoctoral Training in Cardiovascular Physiology, The Ohio State University, 2019-2023

Julie Ledford, PhD

I am an Associate Professor with tenure in the Department of Cellular and Molecular Medicine at the University of Arizona. I am also an active member of the Asthma and Airway Disease Research Center and the BIO5 Institute and have faculty appointments in the Graduate Interdisciplinary Program in the Physiological Sciences and in the Allergy and Immunology Fellowship Training program. I serve as co-director of the Lung Research Focus group (RGF2) within the Southwest Environmental Health Sciences Center.

Darren Cusanovich, PhD

Our lab is interested in understanding how the human genome regulates itself to bring about all of the cellular diversity present in our bodies. In addition, we are interested in how genetic variation and environmental exposures in human populations impact that regulation and sometimes leads to complex disease. The particular disease model that we focus on is asthma, a complex disease affecting ~10-20% of the population that involves many cell types of the lung and immune system and offers exquisitely detailed examples of gene-environment interactions that influence disease outcomes.

Jared Churko, PhD

Jared Churko, PhD, joined the University of Arizona Department of Cellular and Molecular Medicine in April 2018 following his position as an Instructor within Stanford University's Cardiovascular Institute. His lab is located within the Sarver Heart Center, where he studies heart development and cardiovascular disease. Specifically, his lab combines systems biology, stem cell biology, cardiac biology, genetic engineering, and bioinformatics to understand the mechanisms leading to heart disease.

Coen Ottenheijm, PhD

In healthy humans, ventilation is propelled by the rhythmic contraction of the diaphragm, the main muscle of inspiration. In contrast, in patients admitted to the intensive care unit, ventilation is carried out by a machine - to facilitate oxygen uptake in the lungs - and the diaphragm is inactive. However, mechanical ventilation is clearly a two-edged sword: recent studies suggest that during mechanical ventilation the diaphragm rapidly weakens. Thus, prolonged mechanical ventilation may be required because of diaphragm weakness caused by the mechanical ventilation itself.

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